Small Methods. 2024 Jan 2:e2301310.doi: 10.1002/smtd.202301310. Online ahead of print. An Inducible CRISPR-dCas9-Based Transcriptional Repression System for Cancer Therapy 一种用于癌症治疗的基于诱导CRISPR-dCas9的转录抑制系统(IF:25.367).
Autophagy. 2023 Dec 12:1-28.doi: 10.1080/15548627.2023.2293442. Online ahead of print. Senecavirus A induces mitophagy to promote self-replication through direct interaction of 2C protein with K27-linked ubiquitinated TUFM catalyzed by RNF185 Senecavirus A通过RNF185催化2C蛋白与K27连接的泛素化TUFM的直接相互作用诱导线粒体自噬以促进自身复制(IF:13.391).
Signal Transduct Target Ther . 2023 Aug 7;8(1):292. doi: 10.1038/s41392-023-01543-z. Extracellular RNAs-TLR3 signaling contributes to cognitive impairment after chronic neuropathic pain in mice.(IF:39.3).
Sci Adv. 2023 Jul 21;9(29):eadf7858.doi: 10.1126/sciadv.adf7858. Epub 2023 Jul 21. Engineered exosomes reprogram Gli1+ cells in vivo to prevent calcification of vascular grafts and autologous pathological vessels 工程外泌体在体内重新编程Gli1+细胞，以防止血管移植物和自体病理血管钙化 (IF:14.98).
Theranostics. 2023 Jun 26;13(11):3761-3780.doi: 10.7150/thno.83676. eCollection 2023. Sufu limits sepsis-induced lung inflammation via regulating phase separation of TRAF6 苏福通过调节TRAF6的相分离来限制脓毒症诱导的肺部炎症(IF:11.6).
Signal Transduct Target Ther. 2023 Apr 3;8(1):144.doi: 10.1038/s41392-023-01351-5. tRF-Gln-CTG-026 ameliorates liver injury by alleviating global protein synthesis (IF:39.3).( 通过减轻整体蛋白质合成改善肝损伤 )
Cell Mol Immunol . 2022 Sep 1. doi: 10.1038/s41423-022-00917-7. Online ahead of print. USP3 deubiquitinates and stabilizes the adapter protein ASC to regulate inflammasome activation. （体内尾静脉蛋白转染)(IF:22.096).
Research (Wash D C) . 2022 Jul 19;2022:9826426. doi: 10.34133/2022/9826426. eCollection 2022. Programming of Regulatory T Cells In Situ for Nerve Regeneration and Long-Term Patency of Vascular Grafts. (IF:11.036).
Mol Ther.2022 Jan 22;S1525-0016(22)00033-8.doi: 10.1016/j.ymthe.2022.01.033.A microRNA checkpoint for Ca 2+ signaling and overload in acute pancreatitis. (IF:11.454).
Cell Res.2021 Sep 24.doi: 10.1038/s41422-021-00565-y. Nuclear UHRF1 is a gate-keeper of cellular AMPK activity and function (IF:25.617).
